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Katherine Kay Thompson, beloved wife of the late Harry Thompson, passed away at Good Samaritan Society South Ridge Village on Tuesday, January 15th, 2013 at the age of 92 years.
Kay leaves to cherish her memory four sons, Lynn (Susan), Gordon, Sid (Joan), and Barry (MaryJean); three daughters, Linda (Ron) Windrim, Kathy (Dale) Garinger, and Cindy (Terry) Babiy. She was the proud grandmother of eleven grandchildren, Michelle (Jason) Bodnarchuk, Kevin Martin, Jennifer Garinger, Blair Garinger, Craig (Erica) Windrim, Jonathan Babiy, Lindsay (Tyrell) Parenteau, Greg Thompson, Megan Thompson, Mike (Michelle) Long and David Long; and great grandmother to Lyndon and Adrianna Bodnarchuk and Barrett Long. Kay is also survived by her sisters, Violet Weiss and Carrie Schneider; and her son-in-law, Howard Martin; plus numerous nieces and nephews. Kay was predeceased by her husband of 43 years, Harry Thompson (1985); her daughter, Vicky Martin (2007); her parents, Jacob and Magdelena Bender; and her siblings, John, Philip, Jack, Freddie, Helen Frank, Martha Opp, Liz Hughes, Christine Krassman and Lena Deering. Kay was born November 28th, 1920 in Hilda, Alberta to Jacob and Magdelena Bender. She married Harry Thompson on May 11th, 1942. Together they farmed in Rapid City, Manitoba until 1950. When Harry was offered a job working with the Defence Research Establishment Suffield, they relocated to Redcliff and then to Ralston. They moved the family to Medicine Hat in 1963. Kay spent most of her life as a stay-at-home mom raising her eight children. She tended to a big vegetable garden and always had flowers in the front yard. She enjoyed gardening, cooking, baking and family outings -camping and fishing at Lake Newell or Elkwater. She was a huge Blue Jays fan and an avid bingo player. Family was most important to Kay and she was happiest when everyone managed to get together. She will be missed by her family and friends but we can be assured she is now content and with her dear Harry again. Those wishing to pay their respects may do so at SAAMIS MEMORIAL FUNERAL CHAPEL, #1 Dunmore Road SE, on Thursday from 7:00 to 9:00 p.m. and on Friday from 12:00 to 12:55 p.m. The Funeral Service will be held in SAAMIS MEMORIAL FUNERAL CHAPEL on Friday, January 18th, 2013 at 1:00 p.m. with Pastor Garth Wehrfritz-Hanson officiating. Interment will follow in the Family Plot at Saamis Prairie View Cemetery. Memorial gifts in Kays memory may be made to the Good Samaritan Society, 550 Spruce Way SE, Medicine Hat, Alberta, T1B 4P1. Condolences may be sent through www.saamis.com or www.gonebutnotforgotten.ca or to
subject heading Kay Thompson. Radio funeral announcement can be heard on Thursday around 12:35 p.m. on 93.7 Praise FM. Funeral arrangements are entrusted to SAAMIS MEMORIAL FUNERAL CHAPEL AND CREMATORIUM, "The Chapel in the Park", locally owned and directed. Should you wish any additional information, please telephone (403)528-2599
Although you may have lost someone special to you, your memory of them lives on.
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Some authors advocate measuring prolactin and free testosterone levels as part of the initial evaluation.4,5 The value of this routine testing is uncertain, however, because the incidence of endocrinopathy presenting as erectile dysfunction is reportedly only 2 percent.8 A reasonable strategy is to obtain a free testosterone level in all men aged 50 and older and in those younger than 50 who have symptoms or signs of hypogonadism such as decreased libido, bilateral testicular atrophy or a reduced amount of body hair.9 The prolactin level should be measured if the free testosterone level is low, the patient has a substantial loss of libido or a prolactinoma is suspected on the basis of a history of headache with visual field cuts.10 Measurement of luteinizing hormone levels can be reserved for use in distinguishing primary from secondary hypogonadism in men with low testosterone levels.9
Methods and Results— We evaluated the prevalence of ED in 133 uncomplicated diabetic men with angiographically verified silent CAD and in 127 diabetic men without myocardial ischemia at exercise ECG, 48-hour ambulatory ECG, and stress echocardiography. The groups were comparable for age and diabetes duration. Patients were screened for ED using the validated International Index of Erectile Function (IIEF-5) questionnaire. The prevalence of ED was significantly higher in patients with than in those without silent CAD (33.8% versus 4.7%; P=0.000). Multiple logistic regression analysis showed that ED, apolipoprotein(a) polymorphism, smoking, microalbuminuria, HDL, and LDL were significantly associated with silent CAD; among these risk factors, ED appeared to be the most efficient predictor of silent CAD (OR, 14.8; 95% CI, 3.8 to 56.9).
A. While the injections have a high success rate (over 70 percent), the drugs may not be able to override poor blood flow to the penis. In some cases, the trimix may produce an initial erection, but venous drainage (inability of penile tissues to trap blood) in the penis allows blood to escape rapidly, resulting in an unsustainable erection.
According to Israeli research, the same benefits could help men who suffer persistent erection problems.
One of the very frequent disorders that have affected additional than 90% of the sexually active men a lowest possible of once is the impotence.
Key Words: erectile dysfunction, phosphodiesterase inhibitors: sildenafil, tadalafil, vardenafil
Cook noted that this study is unique because, unlike previous studies, it didn't focus on men who obtained...
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Ginkgo biloba – Increases arterial blood flow, which may have a positive effect on male sexual function.
Figure 1. PDE5 inhibition imparts a measurable immune-mediated antitumor effect. BALB/c or BALB/c Rag-2–/– mice were challenged s.c. with 0.5 x 106 CT26WT (A), C26GM colon carcinoma (B), or TS/A mammary adenocarcinoma (C) cells. (D) C57BL/6 mice were challenged with 0.5 x 106 MCA203 s.c. PDE5 inhibitors were started on day 0. Sildenafil was added to the drinking water or given i.p. daily. Tadalafil was given i.p. daily where indicated in B. Error bar values are shown.
Also, surgery (especially radical prostate and bladder surgery for cancer) can injure nerves and arteries near the penis, causing ED. Injury to the penis, spinal cord, prostate, bladder, and pelvis can lead to ED by harming nerves, smooth muscles, arteries, and fibrous tissues of the corpora cavernosa.
"To our knowledge, there are no previous studies on the incidence of erectile dysfunction in relation to NSAID use, i.e. studies on the use of NSAIDs recorded before the occurrence of erectile dysfunction," Shiri's team, from the University of Tampere in Finland, points out.
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