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Wednesday, 23 January 2013 18:03
Evelyn Ethel Litwin passed away peacefully at the Lethbridge Regional Hospital on Saturday, January 19th, 2013 at the age of 78 years.
Evelyn was born on November 26th, 1934 in Basswood, Manitoba, the eldest daughter of William and Ethel Pearl (Minard) Shewfelt.
She was a warm, generous and kind woman who will be remembered and missed lovingly by her family, Darlene (Ken) Hooper and Brian (Ruth) Litwin of Lethbridge and Rose-Marie Litwin of Windsor, Ontario and grandchildren Josh (Megan) of Calgary and Camille (James) of Lethbridge.
She is also survived by one brother Mervin (Val) of Esterhazy, SK and a sister, Joyce Robinson of Regina, SK and brother-in-law and sister-in-law, Gordon and Rose Marie Litwin of Lethbridge.
Also, she will be missed greatly by the many friends and acquaintances she gathered over the years who were charmed by her truly sweet, accepting and uncritical nature. She loved to laugh.
She was predeceased in 1980 by her husband Robert George Litwin.
Inurnment will take place at MOUNTAIN VIEW CEMETERY on Wednesday, January 23rd, 2013, at 11:50 A.M. commencing at the main entrance, to be followed by a Memorial Service at FIRST BAPTIST CHURCH, 1614 5th Avenue South, Lethbridge at 1:00 PM with Pastor Bruce Martin officiating.
Memorial donations in lieu of flowers may be made to the Chinook Regional Hospital Foundation, 960 19th Street South, Lethbridge, T1J 1W5
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Attention has recently been focused on the chemical mediators of erection. These include prostaglandin E, acetylcholine, vasoactive intestinal peptide and nitric oxide. The most physiologically important mediator is nitric oxide.2 This substance is released from nerve endings and vascular endothelium and functions by activating guanylate cyclase, which converts guanosine triphosphate to cyclic guanosine monophosphate (cGMP).3 cGMP becomes the secondary messenger that causes smooth muscle relaxation, resulting in venous engorgement and penile tumescence.
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The workers studied did not have to spend years in the factory to develop problems -- sexual dysfunction began in new workers after just months on the job, Li said.
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"There are several reasons ophthalmologists need to be acquainted with the pharmacologic profiles of PDE5 inhibitors and their potential side effects," the authors wrote "The frequency of erectile dysfunction, which is a form of peripheral vascular disease that impairs men's abilities to achieve and maintain an erection, increases dramatically with age and in the presence of cardiovascular risk factors. Therefore, many men who take PDE5 inhibitors to treat their erectile dysfunction will also be followed up by ophthalmologists for ocular disorders such as diabetic retinopathy, macular degeneration and ocular vascular disease."
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Aging: There are two reasons why older men are more likely to experience erectile dysfunction than younger men. First, older men are more likely to develop diseases (such as heart attacks, angina, strokes, diabetes mellitus, and high blood pressure) that are associated with erectile dysfunction. Second, the aging process alone can cause erectile dysfunction in some men; primarily by decreasing the compliance of the tissues in the corpora cavernosa, although it has been suggested, but not proven, that there is also decreased production of nitric oxide in the nerves that innervate the corporal smooth muscle within the penis.
Erectile dysfunction is most likely the many usual sexual dysfunction among men within the United States with regarding 300,000 instances diagnosed any year in addition to the overwhelming majority of unreported cases.
Figure 5. Sildenafil down-regulates IL-4R expression on CD11b+ cells. CD4+, CD8+, CD11b+, or CD11c+ cells were magnetically purified from spleens of C26GM-bearing mice challenged 9 d earlier. CD4+ or CD8+ T cells were stimulated with ConA for 3 d, with or without 50 µg/ml sildenafil. (A) Purified cells were cultured separately for 2 d with or without sildenafil, harvested, and used to determine cGMP levels, as described in Materials and methods. (B) T cell proliferation was determined by [3H]thymidine incorporation. (C) CD11c+ cells were analyzed for B7.2 and MHC class II expression. (D) CD11b+ cells were analyzed for IL-4R expression by flow cytometry. The t test p-value is reported. Error bar values are shown. MFI, multiplicity of infection.
Figure 7. PDE5 inhibition reverts MDSC suppressive pathways. Splenic CD11b+ cells from C26GM tumor-bearing mice were isolated and added to CFSE-labeled splenocytes containing either (A) naive HA-specific CD8+ (CL4) cells or (B) naive HA-specific CD4+ (6.5) cells. The cultures were stimulated for 4 d with the relevant peptide in the presence or absence of 50 µg/ml sildenafil. Proliferation was evaluated as CFSE dilution by FACS analysis. (C) Splenic CD11b+ cells were magnetically purified from B16GM tumor-bearing C57BL/6-NOS+/+ or C57BL/6-NOS–/– mice and added to CFSE-labeled splenocytes containing naive OVA-specific CD4+ T cells. The cultures were stimulated for 4 d with the relevant peptide in the presence or absence of 50 µg/ml sildenafil. Proliferation was evaluated as CFSE dilution by flow cytometry. Data derived from one of two independent experiments with similar results are reported. Error bar values are shown.
Scientists Create Artificial Penis
Despite considerable recent experimentation in animal models and human volunteers, information on the central pathways of erection remains cursory at best.18 It is known that androgens play a predominantly modulating role by their effect on libido and sexual behaviour. Testosterone enhances sexual interest and the frequency of sexual acts; it increases the frequency of nocturnal erections but does not effect reflexogenic or psychogenic erections.19
These drugs are called phosphodiesterase-5 (PDE5) inhibitors.
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